{"id":34456,"date":"2024-02-05T00:01:31","date_gmt":"2024-02-05T05:01:31","guid":{"rendered":"https:\/\/beyondpesticides.org\/dailynewsblog\/?p=34456"},"modified":"2024-02-05T11:15:42","modified_gmt":"2024-02-05T16:15:42","slug":"epas-proposed-endocrine-disrupting-pesticide-review-called-deficient","status":"publish","type":"post","link":"https:\/\/beyondpesticides.org\/dailynewsblog\/2024\/02\/epas-proposed-endocrine-disrupting-pesticide-review-called-deficient\/","title":{"rendered":"EPA\u2019s Proposed Endocrine Disrupting Pesticide Review Called Deficient"},"content":{"rendered":"\r\n

(Beyond Pesticides<\/em>, February 5, 2024) Public Comment Period Ends February 26, 2024.<\/strong> The U.S. Environmental Protection Agency (EPA) never completed protocol for testing pesticides that disrupt the fundamental functioning of organisms, including humans, causing a range of chronic adverse health effects that defy the common misconception that dose makes the poison (\u201ca little bit won\u2019t hurt you\u201d)\u2014when, in fact, minuscule doses (exposure) wreak havoc with biological systems. After a nearly two decade defiance of a federal mandate<\/a> to institute pesticide registration requirements for endocrine disruptors, EPA has now opened a public comment period ending February 26, 2024<\/a> and advocates are criticizing the agency\u2019s proposed evaluation as too narrow.<\/p>\r\n

A detailed examination of EPA’s proposal can be found in draft comments by Beyond Pesticides<\/a>.<\/em>\u00a0<\/p>\r\n

Endocrine disruption as a phenomenon affecting humans<\/a> and other species<\/a> has been critically reviewed by many authors<\/a>. Endocrine disruptors are chemicals that can, even at extremely low exposure levels, disrupt normal hormonal (endocrine) function. Such endocrine disrupting compounds (EDC) include many pesticides, exposures to which have been linked to infertility and other reproductive disorders, diabetes, cardiovascular disease, obesity, and early puberty, as well as attention deficit hyperactivity disorder (ADHD), Parkinson\u2019s, Alzheimer\u2019s, and childhood and adult cancers. EPA and its Endocrine Disruptor Screening Program (EDSP) began then virtually stopped its review and regulation of endocrine disrupting pesticides, despite a mandate in the 1996 Food Quality Protection Act\/Federal Food, Drug, and Cosmetic Act<\/em> (FQPA\/FFDCA), as well as the Federal Insecticide, Fungicide, and Rodenticide Act<\/em> (FIFRA) and Safe Drinking Water Act<\/em> (SDWA), to develop a screening program within two years and then begin regulating.<\/p>\r\n

>>Tell EPA that it must complete comprehensive data analysis concerning endocrine disruption and not register pesticides without it<\/a>.<\/strong><\/p>\r\n

\u201cBecause of its limited scope, the EPA\u2019s proposed framework for review of endocrine disrupting properties of pesticides is an abrogation of its responsibilities,\u201d said Beyond Pesticides. The limitations in EPA\u2019s proposal cited, despite Congressional intent and statutory mandates, include: (i) applying EDSP to humans only; (ii) focusing on certain pesticide active ingredients only, and; (iii) restricting the types of data used to assess endocrine disruption (ED). The proposal also reverses advice of EPA\u2019s Endocrine Disruptor Screening and Testing Advisory Committee (EDSTAC) and the agency\u2019s original EDSP implementation policy and science decisions.<\/p>\r\n

Under FIFRA, a pesticide is presumed to pose an unreasonable risk until reliable data demonstrate otherwise. Moreover, if the agency lacks the data and\/or resources to fully evaluate endocrine risks to human health and wildlife, then the agency is obliged to suspend or deny any pesticide registration until it has sufficient data to demonstrate that the pesticide\u2019s registration is in compliance with the statutory standard, which is no \u201cunreasonable adverse risk\u201d of endocrine disruption.<\/p>\r\n

According to Beyond Pesticides, EPA cannot develop a strategy for evaluating pesticides without understanding the history and status of endocrine disruption research, which are summarized in Beyond Pesticides\u2019 draft comments<\/a>. Evidence that synthetic chemicals can mimic or otherwise interfere with natural hormones has existed for over half a century. Although early attention was given to estrogen mimics<\/a>, it soon became apparent that the homeostatic (stable) function of the endocrine system\u2014which regulates and balances physiological functions\u2014can be disrupted at many sites and hormone systems<\/a>.<\/p>\r\n

Endocrine pathways are largely conserved (identical or similar) across species and, thus, are not species\u2014or taxa\u2014specific. It is well known that thyroid endocrinology in particular is well conserved across vertebrate taxa. This includes aspects of thyroid hormone synthesis, metabolism, and mechanisms of action. Thyroid hormones are derived from the thyroid gland through regulation of the hypothalamic-pituitary-thyroid (HPT) axis, which is controlled through a complex mechanism of positive and negative feedback regulation. Multiple pathways contribute to the synthesis of thyroid-releasing hormone, including thyroid hormone signaling through feedback mechanisms; leptin and melanocortin signaling; body temperature regulation; and cardiovascular physiology. Each pathway directly targets thyroid-releasing hormone neurons. Based on the similarities sensitivity and requirements of endocrine pathways across vertebrate species, it is well understood that the ecological assays (the frog assay in particular) are often more sensitive and equally relevant to mammalian assays in informing risk assessors of whether a chemical can perturb and cause adverse endocrine outcomes in the human population and vice versa.<\/p>\r\n

FQPA essentially amends FIFRA to ensure potential endocrine disrupting effects are considered in agency risk assessments to fulfill the FIFRA mandate that a pesticide registration will not cause unreasonable adverse effects. This applies to humans and wildlife and to all pesticide chemicals as defined in FIFRA, including \u201call active and pesticide inert ingredients of such pesticide\u201d (21 U.S.C. 231(q)(1)). SDWA adds drinking water contaminants as well.<\/p>\r\n

In summary, the agency cannot limit EDSP to only humans and conventional pesticide active ingredients without violating the statutory requirements enumerated in FIFRA, FQPA, and SDWA. EPA should make use of all available scientifically relevant endocrine disruption research findings and also be wary of deviating from established international efforts for screening\/testing endocrine disruptors that incorporate human and wildlife relevant studies.<\/p>\r\n

Recognizing that mammalian data inform potential endocrine disruption in other vertebrate taxa (avian, amphibian, fish) and vice versa, the agency should not decouple the mammalian from other vertebrate assays in EDSP screening.\u00a0 There are more than 50 different ecological and mammalian assays included in the Organization of Economic Cooperation and Development (OECD) Conceptual Framework<\/a> for screening\/testing endocrine disrupting effects, and there are additional assays being developed for consideration as well. So, the agency should not limit the range or types of data to be used, but, as FQPA prescribes, use \u201cappropriate validated test systems and other scientifically relevant information.\u201d While currently required data may meet the needs of human risk assessment, it is inadequate to evaluate endocrine effects on wildlife species. It should also be understood that under FIFRA, a pesticide is presumed to pose an unreasonable risk until reliable data demonstrate otherwise. If the agency lacks the data and\/or resources to fully evaluate endocrine risks to human health and wildlife, then the agency is obliged to suspend or deny any pesticide registration until the agency has sufficient data to demonstrate no unreasonable adverse endocrine risk per the mandate in FIFRA. Further, it is not the agency but pesticide registrants (manufacturers) that have the burden to demonstrate with adequate data that their products will not<\/u> pose unreasonable adverse effects, including the inherently presumed endocrine disrupting effects.<\/p>\r\n

Submit comments to Regulations.gov before February 26, 2024<\/a>.
\r\n<\/strong>[Document ID # EPA-HQ-OPP-2023-0474-0005]<\/a><\/p>\r\n

Due to updates to the Regulations website, we are now able to offer a click-and-submit form to the Regulations docket! Please\u00a0fill out the form linked to submit<\/a>!<\/strong><\/em><\/p>\r\n

Suggested comments to EPA:
\r\n<\/strong>EPA\u2019s proposed endocrine disruptor strategy (EDSP) abrogates its responsibilities under the Food Quality Protection Act\/Federal Food, Drug, and Cosmetic Act (FQPA\/FFDCA), the Federal Insecticide, Fungicide, and Rodenticide Act (FIFRA), and Safe Drinking Water Act (SDWA). Limiting the scope of the EDSP to humans, certain pesticide active ingredients only, and limiting the types of data to assess endocrine disruption (ED) effects is contrary to the Congressional intent and requirements in these statutes. It is also a reversal of the Endocrine Disruptor Screening and Testing Advisory Committee advice and EPA\u2019s original EDSP implementation policy and science decisions.<\/p>\r\n

EPA\u2019s strategy for evaluating pesticides requires understanding the history and status of endocrine disruption research. Evidence that synthetic chemicals can interfere with natural hormones has existed for over half a century. Although early attention was given to estrogen mimics, it was soon apparent that the homeostatic function of the endocrine system can be disrupted at many sites and hormone systems.<\/p>\r\n

Many authors have documented that endocrine-disrupting chemicals (EDCs) may be pervasive in contaminating the ecosphere. A pandemic of endocrine-related disorders\u2014attention deficit and hyperactivity disorder (ADHD), autism, diabetes, obesity, childhood cancers, testicular cancer in young men, infertility, male dysgenesis syndrome, hypospadias, low sperm count, loss of semen volume and sperm quality, and increased risk of testicular and prostate cancer\u2014may be related to EDCs.<\/p>\r\n

Endocrine pathways are largely conserved across species and thus are not species\u2014or taxa\u2014specific. Therefore, it is well understood that the ecological assays (the frog assay in particular) are often more sensitive and equally relevant to mammalian assays in determining whether a chemical can perturb and cause adverse endocrine outcomes in the human population and vice versa.<\/p>\r\n

In summary, EPA cannot limit EDSP to only humans and conventional pesticide active ingredients without violating the statutory requirements of FIFRA, FQPA, and SDWA. EPA must use all available scientifically relevant endocrine disruption research findings and avoid deviating from established international efforts that incorporate human and wildlife studies.<\/p>\r\n

Recognizing that mammalian data inform potential endocrine disruption in other vertebrates (avian, amphibian, fish) and vice versa, the agency should not decouple the mammalian from other vertebrate assays in EDSP screening. With more than 50 different ecological and mammalian assays included in the Organization of Economic Cooperation and Development Conceptual Framework for screening\/testing endocrine disrupting effects and additional assays in development, EPA must not limit the range or types of data to be used, but as FQPA prescribes, use \u201cappropriate validated test systems and other scientifically relevant information.\u201d Currently required data may meet the needs of human risk assessment but is inadequate to evaluate endocrine effects on wildlife species.<\/p>\r\n

FQPA amends FIFRA to ensure potential endocrine disrupting effects are considered in risk assessments to fulfill the FIFRA mandate that pesticide use will not cause unreasonable adverse effects. This applies to humans and wildlife and to all pesticide chemicals as defined in FIFRA, including all active and inert ingredients. Under FIFRA, a pesticide is presumed to pose an unreasonable risk until reliable data demonstrate otherwise. If EPA lacks the data and\/or resources to fully evaluate endocrine risks to human health and wildlife, then the agency is obliged to suspend or deny any pesticide registration until the agency has sufficient data to demonstrate no unreasonable adverse endocrine risk.<\/p>\r\n

Thank you.<\/p>\r\n

 <\/p>\r\n","protected":false},"excerpt":{"rendered":"

(Beyond Pesticides, February 5, 2024) Public Comment Period Ends February 26, 2024. The U.S. Environmental Protection Agency (EPA) never completed protocol for testing pesticides that disrupt the fundamental functioning of organisms, including humans, causing a range of chronic adverse health effects that defy the common misconception that dose makes the poison (\u201ca little bit won\u2019t hurt you\u201d)\u2014when, in fact, minuscule doses (exposure) wreak havoc with biological systems. After a nearly two decade defiance of a federal mandate to institute pesticide registration requirements for endocrine disruptors, EPA has now opened a public comment period ending February 26, 2024 and advocates are criticizing the agency\u2019s proposed evaluation as too narrow. A detailed examination of EPA’s proposal can be found in draft comments by Beyond Pesticides.\u00a0 Endocrine disruption as a phenomenon affecting humans and other species has been critically reviewed by many authors. Endocrine disruptors are chemicals that can, even at extremely low exposure levels, disrupt normal hormonal (endocrine) function. Such endocrine disrupting compounds (EDC) include many pesticides, exposures to which have been linked to infertility and other reproductive disorders, diabetes, cardiovascular disease, obesity, and early puberty, as well as attention deficit hyperactivity disorder (ADHD), Parkinson\u2019s, Alzheimer\u2019s, and childhood and adult cancers. EPA […]<\/p>\n","protected":false},"author":2,"featured_media":34468,"comment_status":"open","ping_status":"closed","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[267,268,85,354,119,274,145,275,276,1],"tags":[1225,827,770,444,605,784,643,1304,1248,1176,600,1305,792,786],"class_list":["post-34456","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-adhd","category-alzheimerss","category-endocrine-disruption","category-environmental-protection-agency-epa","category-infertility","category-learning-disabilities","category-parkinsons","category-reproductive-health","category-take-action","category-uncategorized","tag-animal-health","tag-chemicals","tag-data","tag-endocrine-disruption","tag-epa","tag-fifra","tag-health","tag-hormones","tag-human-health","tag-pesticide-regulation","tag-pesticides","tag-program","tag-regulation","tag-take-action"],"yoast_head":"\nEPA\u2019s Proposed Endocrine Disrupting Pesticide Review Called Deficient - 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