(Beyond Pesticides, July 10, 2026) An article titled “The science of staying bite-free: Balancing natural essential oils, DEET safety and permethrin,” calls attention to insect management options and the subsequent consequences for health and the environment. “While DEET has long been the go-to standard for warding off bugs, concerns over its strong chemical scent, greasy feel, and ability to dissolve plastic gear and synthetic fabrics have driven a massive surge in alternative options,” the article says. The use of DEET (N,N-diethyl-meta-toluamide) dates back to 1946 when it was developed by the U.S. military and is the most common and widely used active ingredient in commercial insect repellents applied to the skin with a warning to wash off when indoors and take special precautions with use on children. Despite widespread use, long-standing and mounting scientific evidence finds adverse effects with exposure to DEET and other insect repellents like the synthetic pyrethroid permethrin. Health Risks While regulatory bodies like the U.S. Environmental Protection Agency (EPA) claim insect repellents are safe for the general public, research connects DEET and permethrin to a wide array of adverse health effects. Cases of severe reactions, such as neurological symptoms or seizures, have also occurred. Beyond Pesticides’ […]

(Beyond Pesticides, July 1, 2026) A review in the International Journal of Cancer links pesticide exposure, particularly in areas with high agricultural crop density, to increased risks for childhood cancers. The team of researchers from the University of Nebraska-Lincoln and University of Nebraska Medical Center, in analyzing epidemiologic studies published between January 1980 and September 2022, says that “this scoping review affirms that a robust body of epidemiology literature already informs how parental and childhood exposure to environmental chemical exposures can be associated with children’s incidence of pediatric leukemia and brain cancer.” The scientific literature shows that pediatric cancer, which is a significant cause of morbidity and mortality in children, is associated with exposure to residential pesticides, pesticides ingested through drinking water, parental exposure, and in areas with close proximity to agricultural areas where pesticides are used. Background According to the American Childhood Cancer Organization, over 15,000 children in the U.S. are diagnosed with cancer each year, with pediatric cancer as the second leading cause of death in children 5–9 years of age and the third leading cause of death in children ages 10–14. (See here.) In agricultural states, such as Nebraska, Iowa, Minnesota, Kansas, Illinois, Ohio, and Missouri, incidence […]

(Beyond Pesticides, June 30, 2026) A peer-reviewed article, published in Scientific Reports, focuses on the link between exposure to pesticide mixtures and Alzheimer’s Disease (AD) prevalence at the county-level across the United States. Alzheimer’s, a type of dementia that affects memory, thinking, and behavior, accounts for 60-80% of dementia cases. In conducting a novel cross-sectional analysis of data on pesticide application intensity and disease prevalence, the researchers, from the Medical University of South Carolina, are able to identify exposure clusters with significant associations to the occurrence of AD. The strongest positive associations, where AD prevalence increases as pesticide exposure increases, are “observed for a soil fumigation/nematicide system, an herbicide-dominant vegetation control regime, and a neuroactive insecticide system,” the authors note. These findings link pesticide mixtures to increased AD rates. (See the full PDF of the study here.) Study Importance and Background AD is a condition that gradually damages and destroys neurons in the brain, with disproportionate risks across the U.S. in certain geographical areas. (See here and here.) “These spatial patterns suggest that contextual and environmental determinants may contribute to disparities in dementia burden beyond established individual-level risk factors,” the researchers state. They continue, “Although AD dementia is the leading […]

(Beyond Pesticides, June 16, 2026) If there is one take-home message regarding reducing risk of childhood leukemias and brain cancers, it is to avoid exposure to pesticides during pregnancy—especially indoor insecticides such as flea and tick products, including DEET, household plant and commercial pesticide treatments, and proximity to pesticide applications in agriculture. A review by researchers at the University of Nebraska Medical Center and School of Natural Resources in Omaha considered 88 epidemiological papers published between 1980 and 2022 on pediatric cancer and environmental pesticide exposure and found elevated rates of pediatric cancers associated with pesticide exposure. The reviewers assessed the known associations between the risk of childhood leukemia and brain tumors and their or their parents’ exposure to pesticides, pesticide breakdown products and mixtures. They asked how important known exposures in drinking water were to the children’s risk, and whether genetics is a primary influence on cancer development. The researchers found that the risk of childhood brain tumors increased 1.5 times if pest control products were applied during the entire year before conception. High-grade glioma risk was four times higher when pesticides were applied during pregnancy. Prenatal exposure to flea and tick products raised risk, especially for children diagnosed under […]

(Beyond Pesticides, May 18, 2026) As the studies continue to mount on the adverse effects of exposure to low levels of organophosphate insecticides, the calls for banning the chemicals are growing. Beyond Pesticides announced an action to “Tell Congress, FDA, and EPA that it is past time to stop the manufacture and use of all organophosphate pesticides, which damage the nervous system and brain at low levels.” There are alternatives to these chemicals that support productive and profitable farming operations. Defying the often-repeated claim that organophosphate pesticide effects occur only at high doses, a recent study by researchers at University of California, San Diego, and the Fundación Cimas del Ecuador in Quito, Ecuador, establishes for the first time the pattern of adverse developmental effects that low-level exposure has on healthy neurological and brain development in children. It is firmly established that widely used organophosphate pesticides in food production and other sites are severely toxic to a broad range of organisms. In what is known as their “classic” mechanism of action, they inhibit acetylcholinesterase (AChE), an enzyme that breaks down the neurotransmitter acetylcholine (ACh), particularly in neuromuscular junctions in the brain. Organophosphates are nerve agents, originally developed by the German company IG Farben (a […]

(Beyond Pesticides, May 8, 2026)  A study from Ecuador establishes for the first time the developmental pattern of nervous system toxicants—still widely used in agriculture, mosquito control, and landscaping—on healthy neurological and brain development in children. It is firmly established that widely used organophosphate pesticides are severely toxic to a broad range of organisms. In what’s known as their “classic” mechanism of action, they inhibit acetylcholinesterase (AChE), an enzyme that breaks down the neurotransmitter acetylcholine (ACh), particularly in neuromuscular junctions in the brain. Not enough AChE leads to a buildup of ACh in motor neurons. Organophosphates deplete AChE, and an acute dose can paralyze the heart and lung muscles, causing death. Chronic exposures are implicated in numerous neurodegenerative diseases, including Alzheimer’s, Parkinson’s, and amyotrophic lateral sclerosis (ALS). Beyond Pesticides’ Gateway on Pesticide Hazards and Safe Pest Management has detailed information on the organophosphates malathion, chlorpyrifos, diazinon, and others. Acetylcholine and AChE are vital biological chemicals conserved across the animal kingdom, from humans to insects and everything in between. Yet there is no established baseline for normal levels of ACh and AChE in humans. Comparing biomarkers of organochlorine exposure with normal values would be a major step forward in assessing the influence […]

(Beyond Pesticides, May 1, 2026) A study in Psychiatry Research, entitled “Toxic Exposure and Rates of Suicidal Thoughts and Behaviors Among U.S. Military Veterans,” adds to the body of science linking poorer mental health among military members with exposure to environmental contaminants. While previous research focuses on mental health broadly, the current observational cohort study focuses on exposure to toxic substances and suicidal thoughts and behaviors (STBs) through the analysis of data from 248,926 U.S. veterans enrolled in the Million Veteran Program (MVP). In comparing self-reported exposures to nine toxicants, including Agent Orange, chemical/biological weapons, anthrax vaccine, solvents/fuels, petroleum combustion products, lead, other metals, pesticides, and open-air burn pits, and mental health records, the researchers find that military veterans with higher toxic exposures are more likely to have STBs. The authors also report that these associations occur across all major service cohorts (pre-Vietnam, Vietnam, post-Vietnam, Gulf War, and post-9/11 eras). “Our work contributes to growing evidence that veterans who report more toxic exposures are more likely to have STBs, consistent with greater risk for other mental health outcomes (e.g., depression, PTSD [post-traumatic stress disorder]),” the researchers conclude. Importance and Background Military members experience increased health risks with exposure to environmental […]

(Beyond Pesticides, March 27, 2026) In a study of birth outcomes in Arizona, published in the Journal of Exposure Science & Environmental Epidemiology, researchers find that preconception and prenatal exposure to certain carbamates, organophosphates, and pyrethroids increases the risk of lower Apgar scores, a metric used to assess neonatal health at one minute and five minutes after birth. The results reveal that exposure to “several pesticide active ingredients at any point during preconception and/or pregnancy were associated with increased odds of low Apgar scores: the carbamates carbaryl and formetanate hydrochloride; the organophosphates diazinon and tribufos; and the pyrethroid cypermethrin.” This multi-institutional study, led by the University of Arizona with researchers from Harvard Chan School of Public Health and UCLA Fielding School of Public Health, provides novel insights, as it incorporates pesticide exposure over a 15-year period both before conception and throughout pregnancy. “To analyze associations of preconception and prenatal exposures to carbamate, organophosphate, and pyrethroid pesticide classes and 25 individual active ingredients with newborn Apgar scores to evaluate the relationship between these exposures and neonatal health,” the authors explain. They continue: “We used pesticide use registry and birth certificate data from 2006 to 2020, linked as part of the Arizona […]

(Beyond Pesticides, March 12, 2026) The science connecting pesticide exposure to neurotoxicity continues to mount. A study in Discover Toxicology highlights neurotoxic pollutants as significant environmental threats, showcasing the adverse impacts on vertebrates’ neurological health from pesticides, including organophosphates, carbamates, and organochlorines. “These substances disrupt normal neurophysiological functions by impairing neurotransmission, generating oxidative stress, provoking neuroinflammation, and initiating neuronal cell death,” the authors say. They continue, “Such disturbances are linked to cognitive deficits, motor impairments, and abnormal neural development.” Neurological conditions can manifest as headaches, muscle weakness, tremors, paralysis, coordination challenges, vision loss, hallucinations, vertigo, seizures, memory loss, slurred speech, trouble breathing with minimal exertion, and more. The range of adverse effects from low-dose, long-term exposure and low-dose (or subchronic) exposure during developmental phases of life raises serious questions about the adequacy of the regulatory review of pesticides, which focuses on acute high and lethal dose exposure. One study on the neurotoxicity of pesticides, published in Chemosphere, concludes, “New regulatory and preventive measures to mitigate the neurotoxic effects of pesticides are needed.” (See also Daily News.) Even at low concentration, chronic exposure to pesticides and other environmental contaminants “poses serious ecological and health concerns” that occur as these chemicals “bioaccumulate […]

(Beyond Pesticides, January 27, 2026) A three-part study published in Molecular Neurodegeneration draws a connection between Parkinson’s disease (PD) and the organophosphate chlorpyrifos. “Since chlorpyrifos (CPF) exposure has been implicated as a risk factor for PD, we investigated its association to incident PD and if this association is biologically plausible using human, rodent, and zebrafish (ZF) studies,” the researchers state. Together, the three experiments “strongly implicate exposure to CPF as a risk factor for developing PD,” as the authors find that exposure, even at very low concentrations, causes selective toxicity to dopaminergic neurons that are critical for functions of movement, cognition, emotion, and more. The results reveal that in humans, long-term residential exposure is associated with more than a 2.5-fold increased risk of developing PD, while exposure in mice causes “motor impairment, dopaminergic neuron loss, microglial activation, and an increase in pathological α-synuclein (α-syn) [proteins primarily found in the brain].” The researchers continue, “Using ZF, we found that CPF-induced dopaminergic neuron loss was at least partially due to autophagy dysfunction and synuclein accumulation, as knocking down LC3 [a protein chain] recapitulated the dopaminergic neuron loss.” These three studies highlight the association of CPF with increased risks for developing PD, as […]

(Beyond Pesticides, December 23, 2025) A literature review of military personnel reveals broad evidence linking their toxic exposure to poorer mental health outcomes. The review, written by medical professionals and researchers throughout the U.S. and published in Medical Care, analyzes the existing literature on associations between military environmental exposures (MEEs) to contaminants, including pesticides, and mental health (MH) outcomes. “We used evidence mapping methodology to systematically search MEDLINE, Embase, PsycINFO, and PTSDpubs for studies of toxic exposure during military service and psychiatric outcomes, which included psychiatric diagnoses, psychiatric symptoms, and neurocognitive functioning,” the authors explain. The 49 studies in the review, covering chemical exposures for military members, involve chemical munitions from the Gulf War era and Agent Orange from the Vietnam War era that are associated with symptoms of depression, PTSD, and anxiety, among others. “Overall, available evidence suggests that veterans reporting environmental toxic exposures may report relatively high levels of mental health needs,” the researchers report. They continue: “To date, no studies have synthesized the existing literature linking MEEs to MH outcomes. In this review, we systematically organize and describe peer-reviewed literature studying associations between MEEs and MH and neurocognitive outcomes (ie, diagnoses and symptoms). Our review focused on […]

(Beyond Pesticides, August 13, 2025) The data on the adverse effects of the insecticide chlorpyrifos, still widely used in food production, continued to accumulate with the latest being a study published in PLOS One that finds perinatal exposure to the chemical in mice can alter sleeping patterns, lead to brain inflammation (particularly in female individuals), and impact gene expression linked to immune response and epigenetic effects. The adverse health effects are greater overall in female mice than male mice, emphasizing the significance of disproportionate impacts across species. Chlorpyrifos has been a threat to human and ecological health for decades, originally as a general-use pesticide for homes, gardens, and agriculture, and then restricted to most nonresidential uses in 2000. Currently, the chemical’s permitted uses include food and feed crops, golf courses, as a non-structural wood treatment, and adult mosquito control for public health (insect-borne diseases) uses only. According to health and environmental advocates, there is a long history of U.S. Environmental Protection Agency (EPA) failure to adequately protect human and environmental health from chlorpyrifos, which is linked to endocrine disruption, reproductive effects, neurotoxicity, brain, kidney, and liver damage, and birth and developmental effects. It took 21 years after negotiating a stop […]

(Beyond Pesticides, April 23, 2025) Recent reviews of scientific literature, in both Chemosphere and Reports in Public Health, associate Parkinson’s disease (PD), the second most prevalent neurodegenerative disease globally, with pesticide exposure. “Given the pervasive nature of pesticide residues in everyday food consumption and inadequate monitoring of their long-term toxicological impacts, the role of pesticide exposure as a modifiable risk factor for neurological disorders, including PD, warrants urgent attention,” the researchers state in the article in Chemosphere. In describing the history of Parkinson’s and previous research, the authors in Reports in Public Health note that while PD etiology is not fully understood, it is a multifactorial disease. “Hereditary factors are present in approximately 10% of diagnosed cases of Parkinson’s disease, presenting early onset; while the other 90% of cases are categorized as idiopathic or sporadic Parkinson’s disease, occurring in older individuals and may be associated with exposure to environmental agents,” the researchers say. This disease, first described by English physician James Parkinson, M.D. in 1817, involves neurochemical changes that present as “the appearance of cardinal motor symptoms, such as bradykinesia, rigidity, postural instability, and rest tremor, which are essential for the clinical diagnosis of the disease,” the researchers note. The […]

(Beyond Pesticides, February 7, 2025) With the strong associations established in epidemiologic studies and a dearth of evidence on the actual mechanisms by which pesticides inflict their damage on the brain, a new meta-analysis identifies studies that pesticides can overwhelm cells’ defenses against them, interfere with cell communication in the brain, and disrupt the epigenetic (gene function) regulation of gene expression. In the journal Nucleus, Bilal Ahmad Mir, PhD and colleagues at the University of Kashmir in Srinagar, India, review what is known at the molecular level about pesticides’ role in brain cancer. Brain cancer is a heavy burden for its victims and their families, and attributing a cause is, as with many diseases, fraught with difficulty. But there are strong clues: For example, farming is a known risk factor. In a 2021 meta-analysis of epidemiologic studies between 1998 and 2019, analyzed here by Beyond Pesticides, farming was associated with a 13% increase in the risk of brain cancer. For farmers whose specific exposure to pesticides was documented in the studies, the increase jumped to 20 percent. So there is some reason to point a finger at synthetic chemicals. But agricultural workers are not the only ones at risk. According […]

(Beyond Pesticides, January 31, 2025) A recent cross-sectional study in Heliyon highlights the link between sleep disorders in Thai farmers and pesticide exposure. The authors find pesticide exposure as an important risk factor for sleep disorders after surveying 27,334 farmers over the age of 20 who had work experience for at least five years. The importance of sleep health is reflected both physically and mentally, as studies find “sleep deficiency increase[s] mortality and various health complications, including hypertension, obesity and type 2 diabetes, cardiovascular diseases, mood disorders, and neurodegenerative disorder.” Additional studies find that these issues are compounded when sleep health is affected by environmental factors such as pesticide exposure. (See previous Beyond Pesticides’ coverage here and here.) The researchers report: “The study found a positive association of 19 individual pesticides (twelve insecticides, two herbicides, and five fungicides). Some associations demonstrated a dose-response pattern. Additionally, the study revealed that women are at a higher risk of sleep-related issues with pesticide exposure compared to males. These results not only substantiate existing literature but also unveil several new individual pesticides that may impact sleep health.” Focusing on study participants in Thailand, which is “characterized by heavy pesticide use and minimal protective measures, […]

(Beyond Pesticides, December 12, 2024) A literature review of 161 articles in Discover Toxicology finds that pesticides with different mechanisms of action cause memory and learning impairments. These effects are noted in nontarget species including humans. Pesticide “[e]xposure during development, as well as chronic environmental and occupational exposure, can contribute to decreased cognitive performance,” the researchers say. With a focus on organophosphate pesticides, synthetic pyrethroids, and neonicotinoid insecticides, the authors highlight neurological impacts. Both learning and memory are crucial for the survival of many species. “Considering the importance of learning and memory for human and non-human animal behavior and the growing association between pesticide exposure and cognitive impairment, the aim of this review was to describe the studies showing the impact of pesticide exposure on memory and learning abilities in nontarget species, providing evidence of the impact of pesticides in central nervous system function,” the researchers state. The 161 articles included in the review were identified through database searches in PubMed/Medline and Scielo. The authors note, “Inclusion criteria for article selection included all articles published in English between 2015 and 2024 containing original studies in animals or humans with single or multiple pesticides exposure.” The articles consist of 132 preclinical […]

(Beyond Pesticides, November 5, 2024) Published in the journal Frontiers in Toxicology, a recent study uncovers serious flaws in the pesticide registration process at the U.S. Environmental Protection Agency (EPA) with an in-depth evaluation of the agency’s failure to protect the public from the harmful effects of five neonicotinoid (neonic) insecticides—as mandated by the Federal Insecticide, Fungicide, and Rodenticide Act (FIFRA) and amendments, including Food Quality Protection Act (FQPA) of 1996. This coincides with EPA’s ongoing review to renew their approval for the next 15 years (set to be announced in 2025). The report is based on the first comprehensive assessment of unpublished rodent-based Developmental Neurotoxicity (DNT) studies, conducted between 2000-2003 and submitted by pesticide manufacturers as part of the registration process. All five neonicotinoids evaluated—acetamiprid, clothianidin, imidacloprid, thiacloprid, and thiamethoxam—are associated with significant shrinkage of brain tissue at the highest dosage, according to EPA data reports (see acetamiprid, clothianidin, imidacloprid, thiacloprid, and thiamethoxam). However, with little or no data regarding the chemicals’ impacts at low and mid-level dosages, EPA has either failed to find a “No Observed Adverse Effect Level” (NOAEL) or, seemingly at random, set the NOAEL at the mid-level dosage. The evaluation suggests that perinatal exposure to […]

(Beyond Pesticides, August 15, 2024) Adding to research that has linked insecticide disruption of the gut microbial community to the progression of Parkinson’s Disease (PD) in the brain is a recently published study that further examines the gut-brain mechanism at work. A review by neuroscientist Nabanita Ghosh, PhD in Chemical Research in Toxicology—coauthored by Krishnendu Sinha, PhD, molecular toxicologist and applied pharmacologist, and molecular toxicologist Parames C. Sil, PhD—focuses on how pesticide-induced microbial community alterations specifically drive the initiation of PD and the precise mechanism. The study also explores microbiota changes at different stages of PD progression, offering recent findings.     The researchers derive their data from the examination of PD patients, evaluating the “interaction between pesticides and gut bacteria in PD patients, summarizing how pesticides cause imbalances in gut bacteria, the resulting changes, and their overall effects on the PD prognosis.”   The review “looks at how pesticides and gut bacteria separately influence PD development and progression, highlighting the harmful effects of pesticides and changes in gut bacteria.” The team concludes that pesticide exposure is connected to PD onset through “disturbances in gut function and alterations in intestinal microbiota.” However, the “exact role of microbial factors in this connection remains […]

(Beyond Pesticides, July 18, 2024) A recent study in The Journal of Toxicological Sciences shows that a single dose of the neonicotinoid insecticide clothianidin (CLO) induces behavioral abnormalities, predominantly in female mice, throughout key stages of development. In testing mice at various ages, sex-specific changes were identified that highlight not only varied effects on males and females but also how pesticide exposure at a young age can cause lasting impacts throughout adulthood in mammalian species.  The researchers, at the Laboratory of Animal Reproduction and Development at Tohoku University in Japan, “utilized murine [mouse] models to compare the sex-specific differences in behavioral effects following CLO exposure at different developmental stages. [They] orally administered CLO to male and female mice as a single high-dose solution (80 mg/kg) during the postnatal period (2-week-old), adolescence (6-week-old), or maturity (10-week-old), and subsequently evaluated higher brain function.”   As the authors remark, “Most studies on the neurotoxicity of CLO have targeted only males, with limited insights regarding the neurodevelopmental toxicity in females. There are significant sex differences in brain development due to hormonal, genetic, epigenetic, and other sex-specific factors. Moreover, there are also a number of sex-based differences in the prevalence of developmental disorders, such as […]

(Beyond Pesticides, June 27, 2024) Earlier this month, Public Employees for Environmental Responsibility (PEER) filed a lawsuit against the U.S. Environmental Protection Agency (EPA) on behalf of a group of ranchers and farmers in Texas harmed by biosolids contaminated with per- and polyfluoroalkyl substances (PFAS). The plaintiffs charge that their health and livelihoods were severely damaged due to contaminated biosolids leaching from neighboring properties onto their land. Despite EPA’s responsibility under the Clean Water Act (Section 405(d) and 40 CFR Part 503) to identify toxic pollutants in biosolids and regulate them to protect human health and the environment, the agency has not effectively addressed the dangers posed by PFAS in biosolid fertilizers. EPA’s failure has dramatic impacts on farmers as well as the public, who are eating or drinking PFAS-contaminated crops, dairy milk, beef, or other meat products. The shortcomings of federal regulations underscore the urgent need for a shift in how federal and state agencies approach these issues, prioritizing precaution to prevent future harm. The persistence of these legacy or “forever” chemicals in the environment illustrates the severe consequences of a historically lax regulatory framework in the U.S.  The National Association of State Departments of Agriculture (NASDA) has identified […]

(Beyond Pesticides, April 17, 2024) With headlines drawing public attention to the contamination of drinking water after years of federal government neglect, the U.S. Environmental Protection Agency (EPA) announced on April 10 new standards to reduce public exposure to PFAS, or per- and polyfluoroalkyl substances, commonly referred to as “forever chemicals” because of their persistence. EPA has finalized a National Primary Drinking Water Regulation (NPDWR) for six PFAS, including PFOA and PFOS, which EPA has recognized have no safe level of exposure, regulating new chemicals for the first time since the 1996 amendments to the Safe Drinking Water Act (SDWA). PFAS persistence and bioaccumulation in humans, wildlife, and the environment is due to the strength of a resulting fluorine–carbon atom bond. PFAS contamination of drinking water, surface and groundwater, waterways, soils, and the food supply—among other resources—is ubiquitous worldwide. PFAS is used in everyday products, including cookware, clothes, carpets, as an anti-sticking and anti-stain agent, in plastics, machinery, and as a pesticide. The action was welcomed by environmentalists and public health advocates as an important step but left many concerned that any level of exposure to these chemicals is unacceptable and critical of EPA’s ongoing failure to act despite years […]

(Beyond Pesticides, January 19, 2024) A study published in Environmental Health Perspective is one of the first to indicate a link between exposure to the herbicides 2,4-D and glyphosate and the impairment of behavioral performance (i.e., attention/inhibitory control, memory/learning, language, visuospatial processing, and social perception). These adverse effects associated with the neurotoxic impacts of pesticides on behavior have been previously documented. For example, a study in August 2023 finds oral intake (e.g., eating contaminated foods), inhalation, and dermal exposure to glyphosate lowered cognitive function scores, increased the likelihood of severe depressive symptoms, and impaired auditory (hearing) function. Although previous studies find neurotoxic effects from exposure to these herbicides, very few until now have evaluated how this neurotoxic exposure impacts neurotypical behavior among youth (children and teenagers). The ubiquitous use of glyphosate and 2,4-D use in agriculture—which leaves residues of the toxic chemicals in food and in public areas (e.g., parks and walkways) creates a creates a significant risk for exposure. Glyphosate is already implicated in or proven to lead to the development of numerous health anomalies, including cancer, while 2,4-D also has a range of potential hazards, including cancer. Therefore, studies like this help local and government officials make holistic decisions regarding the use […]

(Beyond Pesticides, January 4, 2024) A review published in the Journal of Neurosciences in Rural Practice finds an association between farmers’ pesticide exposure and cognitive impairment. Specifically, farmers suffer from attention deficit, lack of information processing, non-comprehension of verbal cues, slow processing speed, memory loss, sluggishness, speech difficulties, and impaired motor function. Additionally, the risk of adverse effects from exposure increases with time spent around pesticides, like in other occupational (work-related) settings. Although pesticide exposure may not be the only factor involved in cognitive impairment, exposure can work synergistically (together) with other factors, triggering neurotoxicity. Pesticides play various roles in causing or exacerbating adverse health outcomes like neurotoxic effects and chemical damage to the brain. Numerous pesticides impair neurological function, especially for chronically exposed individuals (e.g., farmworkers) or during critical windows of vulnerability and development (e.g., childhood, pregnancy). Mounting evidence over the past years shows that chronic exposure to sublethal (low) levels of pesticides adversely affects the central nervous system (CNS) and neural receptors, such as connections between nerves, the brain, enzymes, and DNA. Specifically, researchers identify agricultural chemical exposure as a cause of many adverse CNS impacts and neurological diseases, including Alzheimer’s, amyotrophic lateral sclerosis (ALS), and Parkinson’s disease. The researchers reviewed scientific articles published in […]